Universitat de Barcelona
2018-05-02T09:25:39Z
2018-05-02T09:25:39Z
2017-07-20
2018-05-02T09:25:39Z
Nearly 90 years ago, Drury and Szent-Györgyi revealed that adenosine produced profound hypotension and bradycardia, and it affected kidney function in mammals [1]. Since then, the physiological role and potential therapeutic use of adenosine have been extensively explored [2,3]. Adenosine is considered a retaliatory metabolite [4], thus increasing oxygen supply and decreasing oxygen consumption.
Article
Published version
English
Adenosina; Receptors neurals; Purines; Batecs cardíacs; Adenosine; Neural receptor; Purines; Heart beat
MDPI
Reproducció del document publicat a: https://doi.org/10.3390/molecules22071220
Molecules, 2017, vol. 22, num. 7, p. 1220
https://doi.org/10.3390/molecules22071220
cc-by (c) Ciruela Alférez, Francisco et al., 2017
http://creativecommons.org/licenses/by/3.0/es
