dc.contributor.author
Ivanova, Saška
dc.contributor.author
Zorzano Olarte, Antonio
dc.date.issued
2019-12-18T14:28:01Z
dc.date.issued
2020-06-24T05:10:29Z
dc.date.issued
2019-06-24
dc.date.issued
2019-12-18T14:02:15Z
dc.identifier
https://hdl.handle.net/2445/146977
dc.description.abstract
The binding of ligands to death receptors elicits distinct outcomes, such as apoptosis, inflammation and necroptosis, depending on the cellular context. We have recently described that the autophagic protein TP53INP2 favors apoptosis upon death receptor signaling and is a potential biomarker of responsiveness to TRAIL treatment.
dc.format
application/pdf
dc.publisher
Taylor and Francis
dc.relation
Versió postprint del document publicat a: http://dx.doi.org/10.1080/23723556.2019.1632687
dc.relation
Molecular & Cellular Oncology, 2019, vol. 6, num. 5, p. e1632687
dc.relation
http://dx.doi.org/10.1080/23723556.2019.1632687
dc.rights
(c) Taylor and Francis, 2019
dc.rights
info:eu-repo/semantics/openAccess
dc.source
Articles publicats en revistes (Bioquímica i Biomedicina Molecular)
dc.subject
Marcadors bioquímics
dc.subject
Biochemical markers
dc.title
TP53INP2 at the crossroad of apoptosis and autophagy in death receptor signaling
dc.type
info:eu-repo/semantics/article
dc.type
info:eu-repo/semantics/acceptedVersion
