<?xml version="1.0" encoding="UTF-8"?><?xml-stylesheet type="text/xsl" href="static/style.xsl"?><OAI-PMH xmlns="http://www.openarchives.org/OAI/2.0/" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xsi:schemaLocation="http://www.openarchives.org/OAI/2.0/ http://www.openarchives.org/OAI/2.0/OAI-PMH.xsd"><responseDate>2026-04-13T03:49:04Z</responseDate><request verb="GetRecord" identifier="oai:www.recercat.cat:2445/126004" metadataPrefix="qdc">https://recercat.cat/oai/request</request><GetRecord><record><header><identifier>oai:recercat.cat:2445/126004</identifier><datestamp>2025-11-20T13:50:28Z</datestamp><setSpec>com_2072_1057</setSpec><setSpec>col_2072_478799</setSpec><setSpec>col_2072_478916</setSpec><setSpec>col_2072_478917</setSpec></header><metadata><qdc:qualifieddc xmlns:qdc="http://dspace.org/qualifieddc/" xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:dcterms="http://purl.org/dc/terms/" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xmlns:doc="http://www.lyncode.com/xoai" xsi:schemaLocation="http://purl.org/dc/elements/1.1/ http://dublincore.org/schemas/xmls/qdc/2006/01/06/dc.xsd http://purl.org/dc/terms/ http://dublincore.org/schemas/xmls/qdc/2006/01/06/dcterms.xsd http://dspace.org/qualifieddc/ http://www.ukoln.ac.uk/metadata/dcmi/xmlschema/qualifieddc.xsd">
   <dc:title>CARM1 and BAF155: an example of how chromatin remodeling factors can be relocalized and contribute to cancer</dc:title>
   <dc:creator>Stefansson, Olafur A.</dc:creator>
   <dc:creator>Esteller, Manel</dc:creator>
   <dc:subject>Càncer</dc:subject>
   <dc:subject>Metàstasi</dc:subject>
   <dc:subject>Cromatina</dc:subject>
   <dc:subject>Cancer</dc:subject>
   <dc:subject>Metastasis</dc:subject>
   <dc:subject>Chromatin</dc:subject>
   <dcterms:abstract>In a recent article, Wang and colleagues reported the discovery of a mechanism by which CARM1 regulates the genomic localization of BAF155 (a SWI/SNF subunit involved in chromatin remodeling) through post-translational methylation at R1064 arginine residues. This modification leads to the relocalization of BAF155-containing SWI/SNF complexes to regions containing genes involved in the Myc oncogenic pathway. The results presented are evidence that these interactions constitute a mechanism by which the BAF155 chromatin remodeling factor contributes to cancer.</dcterms:abstract>
   <dcterms:issued>2018-11-12T09:34:52Z</dcterms:issued>
   <dcterms:issued>2018-11-12T09:34:52Z</dcterms:issued>
   <dcterms:issued>2014-05-19</dcterms:issued>
   <dcterms:issued>2018-11-12T09:34:52Z</dcterms:issued>
   <dc:type>info:eu-repo/semantics/article</dc:type>
   <dc:type>info:eu-repo/semantics/publishedVersion</dc:type>
   <dc:relation>Reproducció del document publicat a: https://doi.org/10.1186/bcr3657</dc:relation>
   <dc:relation>Breast Cancer Research, 2014, vol. 16, num. 3, p. 307</dc:relation>
   <dc:relation>https://doi.org/10.1186/bcr3657</dc:relation>
   <dc:rights>cc-by (c) Stefansson, Olafur A. et al., 2014</dc:rights>
   <dc:rights>http://creativecommons.org/licenses/by/3.0/es</dc:rights>
   <dc:rights>info:eu-repo/semantics/openAccess</dc:rights>
   <dc:publisher>BioMed Central</dc:publisher>
   <dc:source>Articles publicats en revistes (Ciències Fisiològiques)</dc:source>
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