Calcium alternans produced by increased sarcoplasmic reticulum refractoriness

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dc.contributor	Universitat Politècnica de Catalunya. Departament de Física Aplicada
dc.contributor	Universitat Politècnica de Catalunya. GICITED - Grup Interdiciplinari de Ciència i Tecnologia en l'Edificació
dc.contributor	Universitat Politècnica de Catalunya. NOLIN - Física No-Lineal i Sistemes Fora de l'Equilibri
dc.contributor.author	Rodríguez Cantalapiedra, Inma
dc.contributor.author	Lugo, Carlos
dc.contributor.author	Peñaranda Ayllón, Angelina
dc.contributor.author	Echebarría Domínguez, Blas
dc.date	2011
dc.identifier.citation	Cantalapiedra, I. R. [et al.]. Calcium alternans produced by increased sarcoplasmic reticulum refractoriness. A: Computing in Cardiology. "Proceedings: computing in cardiology". Hangzhou: IEEE Computer Society Publications, 2011, p. 645-648.
dc.identifier.uri	http://hdl.handle.net/2117/16016
dc.language.iso	eng
dc.publisher	IEEE Computer Society Publications
dc.relation	http://cataleg.upc.edu/record=b1383765~S1*cat
dc.rights	Attribution-NonCommercial-NoDerivs 3.0 Spain
dc.rights	info:eu-repo/semantics/openAccess
dc.rights	http://creativecommons.org/licenses/by-nc-nd/3.0/es/
dc.subject	Àrees temàtiques de la UPC::Enginyeria biomèdica::Biomaterials
dc.subject	Àrees temàtiques de la UPC::Física
dc.subject	Intracellular calcium
dc.subject	Electrocardiography
dc.title	Calcium alternans produced by increased sarcoplasmic reticulum refractoriness
dc.type	info:eu-repo/semantics/publishedVersion
dc.type	info:eu-repo/semantics/conferenceObject
dc.description.abstract	Despite the important role of electro-mechanical alternans in cardiac arrhythmogenesis, its molecular origin is not well understood. In this paper we want to study alternans that can be associated with alternations in the cytosolic calcium transient. To this end we study particularly, how a malfunction of Ryanodine Receptor (RyR) recovery time from inactivation may induce beatto- beat alternations in intracellular calcium concentration and how this could affect the stimulus propagation in inhomogeneous tissue, whose cells exhibit different calcium dynamics.

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